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primary antibody against cd206  (Proteintech)
96
Proteintech primary antibody against cd206
ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and <t>CD206</t> (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001
Primary Antibody Against Cd206, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+cd206/pmc12918416-120-0-4?v=Proteintech
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primary antibody against cd206 - by Bioz Stars, 2026-07
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primary antibodies against cd206  (Proteintech)
96
Proteintech primary antibodies against cd206
ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and <t>CD206</t> (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001
Primary Antibodies Against Cd206, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+cd206/pm41480959-252-5-10?v=Proteintech
Average 96 stars, based on 1 article reviews
primary antibodies against cd206 - by Bioz Stars, 2026-07
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primary antibodies against inos  (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc primary antibodies against inos
ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and <t>CD206</t> (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001
Primary Antibodies Against Inos, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+cd206/10__1016_slash_j__colsurfb__2025__115254-217-12-21?v=Cell+Signaling+Technology+Inc
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primary antibodies against inos - by Bioz Stars, 2026-07
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primary antibodies against cd206, na+-k+ atpase, arg-1  (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc primary antibodies against cd206, na+-k+ atpase, arg-1
ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and <t>CD206</t> (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001
Primary Antibodies Against Cd206, Na+ K+ Atpase, Arg 1, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+cd206/pm40611375-297-25-26?v=Cell+Signaling+Technology+Inc
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primary antibodies against cd206, na+-k+ atpase, arg-1 - by Bioz Stars, 2026-07
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primary antibodies against cd206 #24595  (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc primary antibodies against cd206 #24595
ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and <t>CD206</t> (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001
Primary Antibodies Against Cd206 #24595, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+cd206/pm40643547-108-6-13?v=Cell+Signaling+Technology+Inc
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primary antibodies against cd206 #24595 - by Bioz Stars, 2026-07
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primary antibodies against cd206 and inos  (Beyotime)
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Beyotime primary antibodies against cd206 and inos
ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and <t>CD206</t> (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001
Primary Antibodies Against Cd206 And Inos, supplied by Beyotime, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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primary antibodies against cd206 and inos - by Bioz Stars, 2026-07
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primary antibodies against mouse cd206  (Cell Signaling Technology Inc)
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Effects of mAF treatment on hepatic macrophages in CGI-58 LivKO mice. (A) Representative IHC images and quantification of the (B) M1 macrophage marker <t>CD68,</t> (C) M2 macrophage marker CD206, and (D) ratio of <t>CD68</t> to CD206 in the liver of mice. Data are presented as mean ± SD. n = 8 for treatment group, and n = 3 for ALB- group. *P < 0.05, **P < 0.01 by Student’s t-test. Scale bars: 50 μm.
Primary Antibodies Against Mouse Cd206, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+cd206/pmc12179120-149-3-30?v=Cell+Signaling+Technology+Inc
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primary antibodies against cd206 18704–1-ap  (Proteintech)
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Effects of mAF treatment on hepatic macrophages in CGI-58 LivKO mice. (A) Representative IHC images and quantification of the (B) M1 macrophage marker <t>CD68,</t> (C) M2 macrophage marker CD206, and (D) ratio of <t>CD68</t> to CD206 in the liver of mice. Data are presented as mean ± SD. n = 8 for treatment group, and n = 3 for ALB- group. *P < 0.05, **P < 0.01 by Student’s t-test. Scale bars: 50 μm.
Primary Antibodies Against Cd206 18704–1 Ap, supplied by Proteintech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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primary antibodies against cd206  (Cell Signaling Technology Inc)
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Cell Signaling Technology Inc primary antibodies against cd206
Effects of mAF treatment on hepatic macrophages in CGI-58 LivKO mice. (A) Representative IHC images and quantification of the (B) M1 macrophage marker <t>CD68,</t> (C) M2 macrophage marker CD206, and (D) ratio of <t>CD68</t> to CD206 in the liver of mice. Data are presented as mean ± SD. n = 8 for treatment group, and n = 3 for ALB- group. *P < 0.05, **P < 0.01 by Student’s t-test. Scale bars: 50 μm.
Primary Antibodies Against Cd206, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and CD206 (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001

Journal: Hereditas

Article Title: Ferroptosis-dependent small extracellular vesicles ULK1 enhances mitophagy and suppresses breast cancer migration

doi: 10.1186/s41065-025-00621-2

Figure Lengend Snippet: ULK1 in Fer-sEVs inhibits M2 polarization of macrophages and suppresses breast cancer cell migration through mitophagy. A Mitophagy analysis using mCherry-GFP-LC3B tandem fluorescence reporters in macrophages with ULK1 knockdown following Fer-sEVs treatment. B Western blot analysis of macrophage polarization markers of CD86 (M1 marker) and CD206 (M2 marker) after ULK1 knockdown and autophagy induction. C Immunofluorescence staining of CD206 in macrophages after treatment with siNC- Fer-sEVs or siULK1-Fer-sEVs, with or without the autophagy inducer rapamycin. CD206 expression was increased in the siULK1-Fer-sEVs group and suppressed by rapamycin treatment. Scale bar: 50 μm. D Flow cytometric analysis of CD206⁺ macrophages. ULK1 knockdown in Fer-sEVs significantly increased the proportion of CD206⁺ cells, which was reversed by autophagy activation. E Transwell migration assay of MDA-MB-231 cells co-cultured with macrophages pretreated with different sEVs groups. Fer-sEVs-treated macrophages inhibited tumor cell migration, whereas ULK1-deficient Fer-sEVs-treated macrophages restored pro-migratory effects. Scale bar: 50 μm. Data are presented as mean ± SD; ns p > 0.05, * p < 0.05, ** p < 0.01, *** p < 0.001

Article Snippet: Primary antibody against CD206 (Proteintech, 18704-1-AP) was diluted 1:500 in PBS and incubated with cells overnight at 4 °C.

Techniques: Migration, Fluorescence, Knockdown, Western Blot, Marker, Immunofluorescence, Staining, Expressing, Activation Assay, Transwell Migration Assay, Cell Culture

Effects of mAF treatment on hepatic macrophages in CGI-58 LivKO mice. (A) Representative IHC images and quantification of the (B) M1 macrophage marker CD68, (C) M2 macrophage marker CD206, and (D) ratio of CD68 to CD206 in the liver of mice. Data are presented as mean ± SD. n = 8 for treatment group, and n = 3 for ALB- group. *P < 0.05, **P < 0.01 by Student’s t-test. Scale bars: 50 μm.

Journal: Frontiers in Bioengineering and Biotechnology

Article Title: An engineered adipose formulation decreases hepatic inflammation and fibrosis in a rodent model of metabolic dysfunction-associated steatotic liver disease

doi: 10.3389/fbioe.2025.1579062

Figure Lengend Snippet: Effects of mAF treatment on hepatic macrophages in CGI-58 LivKO mice. (A) Representative IHC images and quantification of the (B) M1 macrophage marker CD68, (C) M2 macrophage marker CD206, and (D) ratio of CD68 to CD206 in the liver of mice. Data are presented as mean ± SD. n = 8 for treatment group, and n = 3 for ALB- group. *P < 0.05, **P < 0.01 by Student’s t-test. Scale bars: 50 μm.

Article Snippet: The expression of CD68 and CD206 in the liver was evaluated by immunohistochemical staining using primary antibodies against mouse CD68 (1:4,000 dilution, Abcam, Waltham, MA) or mouse CD206 (1:1,600 dilution, Cell Signaling Technology ® , Danvers, MA).

Techniques: Marker